When an infection is latent, it's normally described as asymptomatic. But there is some reason to think, it might subtly affect humans neurologically in a similar way to how it seems to affect other mammals, like the wolves in this article. But this is controversial.
The behavioral changes are a different story.
I use immunosuppressive medication to manage a chronic condition, so I assume my immune function is worse than someone not using such, but better than e.g. someone undergoing chemotherapy.
Does this make me immunocompromised or not? Or, where does it place me on the scale? What precautions should I take?
It would be useful to understand how your medication interacts with this process.
https://m.youtube.com/watch?v=lXfEK8G8CUI
Somatic hypermutation has a small discussion in the Kerzegast book.
I understand that somatic hypermutation, by the removal of uracil, plays two roles, first in initial recognition of a pathogen, then second in tuning the response for variants.
As I understand it, this only happens in germinal centers, the lymph nodes.
https://www.scribd.com/document/664375598/IMMUNE-by-Philipp-...
This is a question for an infectious disease specialist.
Direct testing for the parasite's DNA in the cerebral-spinal fluid (yeah, a spinal tap sorry) is sensitive, too. Attempting to detect it elsewhere (blood, etc.) works but isn't as reliable. This is more expensive but sometimes used when unsure. Otherwise, in combination with that, it's diagnosed symptomatically. It shows up on a brain scan if the lesions get big enough, not to be alarmist.