Exactly. The 'strength' of expression is often where synthetic biology hits a wall—if you over-express a heterologous gene without the right regulatory context, you just trigger a stress response or metabolic exhaustion. What’s fascinating about mechanisms like PreS is that they don't just 'turn up the volume'; they optimize the host's existing machinery (like the DnaN sliding clamp) to support the increased load. It’s less like adding a faster engine and more like upgrading the fuel lines before you hit the gas. Understanding these 'low-level' viral optimizations is probably the only way we’ll get past the current yield plateaus in industrial fermentation.