Damage to the lung tissue!

There is also a triple anticoagulant therapy of clopidogrel, aspiring, and apixaban. Those studies are popular in South Africa.

Really, thanks a lot, it's a damn burden when your cardiovascular system is feeble, you're constantly on the edge of a problem and have to monitor every effort (almost requiring a car for groceries because i'm not sure I can walk with a backpack multiple times a week).

ps: I've recently learned about the blood viscosity aspect and also red blood cell renewal through "blood donation", you red blood cells being more elastic, it's supposed to ensure better flow in microvasculature.

I have to ask because sometimes you don’t know the credentials and there is a lot of fluff articles and information around.

Pretty interesting that you were able to beat long covid, so you think your regimen helped speed up recovery?

There is a lot of fluff in general, but experience is the best teacher. I'm convinced personally that it helped speed up my recovery, but then again who knows if it is gone for good. That's the vicissitudes of life for ya.

With the caveat that this doesn't contain an answer, cf. cited studies https://www.ncbi.nlm.nih.gov/pmc/articles/PMC8469745/ ("inflammation, difficulty in gas exchange in the lungs and thrombosis" during early infection causes a self-reinforcing cycle of metabolic acidosis: decrease in blood pH leads to decrease in oxygen saturation, which exacerbates the acidosis) and https://pubmed.ncbi.nlm.nih.gov/32738466/ (increased general COVID-19 vulnerability correlating with increased lactate dehydrogenase levels).

The OP study suggests in the broader context that a person more prone to acidosis is more likely to have a much longer and worse experience with COVID. This isn't necessarily news because acidosis was already well correlated with worse short-term COVID outcomes, but if treating the acidosis can substantially treat the symptoms of PASC/long COVID - which one of the authors attempts to experimentally prove on the smallest meaningful sample size of herself - then identifying and treating the cause of _pre-infection_ acidosis risk factors might reduce the likelihood and potency of long COVID.

COVID's already been shown to have a harmful effect on renal health post-illness: https://journals.lww.com/jasn/fulltext/2021/11000/kidney_out..., https://www.nature.com/articles/s41581-021-00487-3

And a strong correlation to both complications in diabetic patients and as a potential cause of diabetes: https://www.cdc.gov/diabetes/library/reports/reportcard/diab...

And since two of the strongest causes of metabolic acidosis are poor renal health and poorly managed diabetes, it feels like there's a case for at least experimentally trying to treat long COVID with a combination of renal nutrition (low-acid/high-alkaline diet, more alkaline-forming foods, alkaline water, less alcohol, more sodium citrate or bicarbonate) and pre-diabetic treatment (stop smoking, exercise, increase low-starch vegetable intake, treat high blood pressure).

Or tl;dr: eat all brussels sprouts and carrots, go play outside more, and stop eating and drinking so much crap. Preferably before you get COVID, but especially after.

Wasn't there something around Cialis / Viagra being explored for covid recovery? Since those drugs act on that, maybe there's a potential connection?

Pushing through is part of the problem. The crash that comes from pushing is the real problem --all kinds of blood markers go amuck after exertion (especially anything aerobic >10m, like a cardio pulmonary exercise test) and stay amuck for about a week. And the more you try to do during the crash the longer or worse the crash becomes. Pushing regardless of crash status, like in graded exercise therapy, has actually caused ME/CFS/Long Covid baseline symptoms to become worse semi-permanently.