The reason why thalidomide was never stopped before approval is because a fetus is only susceptible during a certain growth phase. And in rats (where the teratogenicity was tested) that period is hours, not weeks like humans.
We know how mRNA works. We know what protein is being coded. We know that it codes that protein correctly. We know that the mRNA flushes when it's done. I'm not asking for a smoking gun. I'm asking you to substantiate even the possibility of it with actual peer-reviewed science and not hyperventilating, middlebrow fearmongering.
Bring something real to the table. For once. Any of you.
Again, would such a mechanism have been possible to show for thalidomide? Can you point to any peer-reviewed science that shows any danger of thalidomide, that's dated before the birth defects actually started happening? If your standard of unsafety couldn't be met for something that we now know for sure is unsafe, it's obviously a bad standard to use.
And as a good example, please explain why COX-2 inhibitors increase the risk of heart attacks? Oh, you can't? That's because we don't know the mechanism, but have data to prove they are harmful.
mRNA isn't new and we do know how it works. There is no mRNA left after the process. It breaks down and is evicted. If what you describe was going to happen, it would have happened already.
If you can find anyone who has studied mRNA academically (let's draw a line of "has contributed to or reviewed a paper on the topic", I can't think of a better one?) willing to put their credentials on the line to buttress this concern I'd be honestly amazed, because I can't find one and I have looked.